Berlin, Germany.
-- In trying to counter the worldwide epidemic of Necro-Mortosis, scientists have undertaken a formidable task. The 'Necro virus' has swiftly become the most extensively studied and yet least understood viruses the world has known. Perhaps because it is also one of the most adaptable and elusive.

The key to preventing further widespread growth of the plague is in trying to unlock the genetic code of the virus itself, therefore gaining a greater understanding of how the virus enters human cells.

UCLA Professor David Whister recently released a paper in the Scientific Journal of Medicine stating that: 'all virus's consist of a package of genetic material surrounded by a protein and lipid shell. The type A Necro-Mortosis virus consists of 7 proteins and eight strands of ribonucleic acid (RNA), which carry the code for making the proteins.'

To invade a host, the virus shell includes specific proteins that bind to receptors on the outside of red corpuscles. This is how the virus enters the blood stream. The virus is not air borne and cannot bind to air passages (as first thought). The act of binding draws the virus into the cell membrane. It then fuses and moves through it, emerging into the cytoplasm of the cell. Once there, the shell opens, releasing the ribonucleic acid inside.

The virus then travels quickly throughout the body. Reaching all major organs, central nervous system and brain. Once inside the cerebral cortex, the virus attacks the axons which connect neurons. The axons are surrounded by a fatty insulating sheath called myelin, this is used as an energy source for the virus. It 'feeds' on the myelin. This is what a growing number of scientists now believe tricks the brain to 'believe it's body has actually died.' The body then goes through a protracted state of shock, followed by a slow and painful onset of mortification and necrosis. Death occurs within 4-48 hours.

However, the brain is still alive. The virus in essence, 'tricks' the brain into killing it's own body. But not letting the brain die. The virus feeds nutrients and stimulation to the surrounded brains myelin coating. Thus suspending atrophy of the body, and 're-animating' the host.

The host then needs to gain additional nutrients to continue the feeding of the virus. And so it impulsively searches for food. The source of nutrition required by the virus, aphion A and betax B, can only be found in warm blood and meat. Hence the cycle begins: The hosts needs to kill to serve the virus to keep the host alive.

Studies in Berlin, Germany are showing promising results. Antiviral test drugs can interrupt the process by which a virus reproduces at several stages. The inhibitor XL-6, for example, prevents the virus particle from opening after it enters the cell and can inhibit the manufacture of virus proteins. It also stop the virus from exiting the cell. Although a lot of money, time and hope has been invested in this new drug, XL-6 is still a long way from being fully developed.

For a magnification photo of the virus: Click here

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